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Dr. Bob is Robert Hsiung, MD,
bob@dr-bob.orgShown: posts 1 to 3 of 3. This is the beginning of the thread.
Posted by rianny on November 24, 2003, at 20:34:44
A few weeks ago, I posted a message saying that I think Vitamin B complex I'm taking is worsening my anxiety. Now, I found out that it was Vitamin C that was causing it. The Vita B complex also contained Vita C. In fack, the Vita B complex I have gone thru all contained Vita C.
Just to experiment, I took only Vita C. I have tried two forms of Vita C, one in tablet and another in effervescent. A few moment after taking Vita C, I noticed my body becoming cold and being fatigued.
Does anyone have similar experience or have read about this? I haven't read anything that says Vita C causing anything other than diarrhea as its side effect. This's very strange. Anyway, now I know Vita C makes me sick.
Posted by rianny on November 25, 2003, at 1:44:19
In reply to Vitamin C causing anxiety - It was not Vitamin B, posted by rianny on November 24, 2003, at 20:34:44
Well, maybe I took too much of it. RDA for Vita. C is 60mg, but I took 500mg-1000mg. I'll keep posting after experimenting.
Posted by Larry Hoover on November 25, 2003, at 6:30:33
In reply to Re: Vitamin C causing anxiety - It was not Vitamin B, posted by rianny on November 25, 2003, at 1:44:19
> Well, maybe I took too much of it. RDA for Vita. C is 60mg, but I took 500mg-1000mg. I'll keep posting after experimenting.
You could safely take 10 times what you took, without any risk of harm (except diarrhea).
I'm not trying to argue with you about your experience (I trust your observation about anxiety after ascorbic acid), but it is unanticipated.
Biol Psychiatry. 2002 Aug 15;52(4):371-4.
High-dose ascorbic acid increases intercourse frequency and improves mood: a randomized controlled clinical trial.Brody S.
Center for and the Psychosomatic and Psychobiological Research, University of Trier, Germany.
BACKGROUND: Ascorbic acid (AA) modulates catecholaminergic activity, decreases stress reactivity, approach anxiety and prolactin release, improves vascular function, and increases oxytocin release. These processes are relevant to sexual behavior and mood. METHODS: In this randomized double-blind, placebo-controlled 14 day trial of sustained-release AA (42 healthy young adults; 3000 mg/day Cetebe) and placebo (39 healthy young adults), subjects with partners recorded penile-vaginal intercourse (FSI), noncoital partner sex, and masturbation in daily diaries, and also completed the Beck Depression Inventory before and after the trial. RESULTS: The AA group reported greater FSI (but, as hypothesized, not other sexual behavior) frequency, an effect most prominent in subjects not cohabiting with their sexual partner, and in women. The AA but not placebo group also experienced a decrease in Beck Depression scores. CONCLUSIONS: AA appears to increase FSI, and the differential benefit to noncohabitants suggests that a central activation or disinhibition, rather than peripheral mechanism may be responsible.
This is the end of the thread.
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